Inicio > LOPEZ LOPEZ, JOSE RAMON

LOPEZ LOPEZ, JOSE RAMON

CATEDRATICOS DE UNIVERSIDAD
Bioquímica y Biología Molecular y Fisiología
Edición Génica para el Estudio de Canales Iónicos Vasculares y Proteínas Mitocondriales
 
jrlopez@uva.es

Índice H en Scopus: 32
Experto
  • Bioinstrumentación
  • Cultivo celular
  • Cultivo de tejidos
  • Hipertensión
  • Electrofisiología
  • Fisiología celular
  • Animales de laboratorio
 
DESPACHO 150.
HORARIO DE ATENCIÓN AL ALUMNO: LUNES A VIERNES 9-14.

[][com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=74170]], camposKey:0021-9541 2023-01-01 Characterization of endogenous Kv1.3 channel isoforms in T cells00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=73013]], camposKey:1664-042X 2022-09-23 Vascular smooth muscle ion channels in essential hypertension00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=74169]], camposKey:1664-042X 2022-01-01 Vascular smooth muscle ion channels in essential hypertension00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=63338]], camposKey:1420-682X 2021-12-01 Lipin-1-derived diacylglycerol activates intracellular TRPC3 which is critical for inflammatory signaling82438260, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=65513]], camposKey:2212-8778 2021-11-01 miR-126 contributes to the epigenetic signature of diabetic vascular smooth muscle and enhances antirestenosis effects of Kv1.3 blockers00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=63337]], camposKey:0021-9541 2021-06-01 Voltage-dependent conformational changes of Kv1.3 channels activate cell proliferation43304347, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=73014]], camposKey:2633-8823 2021-01-01 Kv1.3 Channel Inhibition Limits Uremia-Induced Calcification in Mouse and Human Vascular Smooth Muscle00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=58614]], camposKey:1742-7061 2020-10-01 Elastin-like recombinamer-based devices releasing Kv1.3 blockers for the prevention of intimal hyperplasia: An in vitro and in vivo study264274, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=63339]], camposKey:1931-5244 2020-10-01 Kv1.3 blockade inhibits proliferation of vascular smooth muscle cells in vitro and intimal hyperplasia in vivo4054, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=51867]], camposKey:0022-2828 2019-01-01 Activation of the Cation Channel TRPM3 in Perivascular Nerves Induces Vasodilation of Resistance Arteries219230, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=51866]], camposKey:1079-5642 2019-01-01 Myocardin-Dependent Kv1.5 Channel Expression Prevents Phenotypic Modulation of Human Vessels in Organ Culture00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=11841]], camposKey:1073-9688 2018-01-01 Kv channels and vascular smooth muscle cell proliferation00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=11326]], camposKey:1664-042X 2018-01-01 Phenotypic modulation of cultured primary human aortic vascular smooth muscle cells by uremic serum00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=38526]], camposKey:0363-6143 2018-01-01 The secret life of ion channels: Kv1.3 potassium channels and proliferation00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=49457]], camposKey:0006-3495 2018-01-01 Voltage-Dependent Conformational Changes of KV1.3 Potassium Channels are an Essential Element for KV1.3-induced cell proliferation00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=9151]], camposKey:0006-3495 2017-01-01 Activation of TRPM3 in Perivascular Sensory Nerves Induces Dilation of Mouse Resistance Arteries00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=3275]], camposKey:0022-3751 2017-01-01 Differences in TRPC3 and TRPC6 channels assembly in mesenteric vascular smooth muscle cells in essential hypertension14971513, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=9154]], camposKey:0892-6638 2017-01-01 Kv1.3 and Kv1.5 Channels as Novel Targets for the Prevention of Restenosis in Human Vessels00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=9152]], camposKey:1742-7835 2017-01-01 Pharmacological and genetic dissection of the role of TRPA1 and TRPM8 channels in intrinsic vascular responses to cold5858, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=3274]], camposKey:1664-042X 2017-01-01 Proliferative role of Kv11 channels in murine arteries00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=9153]], camposKey:0892-6638 2017-01-01 Purinergic signalling in mesenteric vascular smooth muscle in a mice model of essential hypertension00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2778]], camposKey:0021-9258 2016-01-01 Molecular Determinants of Kv1.3 Potassium Channels-induced Proliferation35693580, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2743]], camposKey:0022-1295 2016-01-01 TRPC Channel Remodeling in a Mouse Model of Essential Hypertension: The TRPC3-TRPC6 Game of Thrones00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2741]], camposKey:0006-3495 2016-01-01 Voltage-Dependent Accessibility of Phosphorylation Sites at the Carboxy-Terminal Domain of Kv1.3 Channels Determines Kv1.3-Induced Cell Proliferation00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2788]], camposKey:0920-3206 2015-01-01 Erratum to: K+ Channels Expression in Hypertension After Arterial Injury, and Effect of Selective Kv1.3 Blockade with PAP-1 on Intimal Hyperplasia Formation [Cardiovascular Drugs and Therapy, 28, (2014), 501-511, DOI:10.1007/s10557-014-6554-5]199200, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2777]], camposKey:0031-6768 2015-01-01 Kv1.3 channels modulate human vascular smooth muscle cells proliferation independently of mTOR signaling pathway17111722, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=52743]], camposKey:0735-1097 2015-01-01 ROLE OF KV1.3 CHANNEL IN VASCULAR SMOOTH MUSCLE CELLS PROLIFERATION IN A PORCINE MODEL OF RESTENOSIS00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2789]], camposKey:1932-6203 2015-01-01 Tungstate-Targeting of BK alpha beta(1) Channels Tunes ERK Phosphorylation and Cell Proliferation in Human Vascular Smooth Muscle121, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2783]], camposKey:0031-6768 2014-01-01 Cinnamaldehyde inhibits L-type calcium channels in mouse ventricular cardiomyocytes and vascular smooth muscle cells20892099, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2787]], camposKey:0920-3206 2014-01-01 K+ Channels Expression in Hypertension After Arterial Injury, and Effect of Selective Kv1.3 Blockade with PAP-1 on Intimal Hyperplasia Formation501511, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2759]], camposKey:1018-1172 2014-01-01 Rediscovering the interplay between Ca2+ and K+ channels in essential hypertension153153, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2775]], camposKey:0379-864X 2014-01-01 TRPA1 channels are neuronal chemosensors of bacterial endotoxins7475, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2790]], camposKey:2041-1723 2014-01-01 TRPA1 channels mediate acute neurogenic inflammation and pain produced by bacterial endotoxins116, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2781]], camposKey:0022-3751 2013-01-01 Down-regulation of Ca(V)1.2 channels during hypertension: how fewer Ca(V)1.2 channels allow more Ca2+ into hypertensive arterial smooth muscle61756191, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=63340]], camposKey:0019-2805 2012-09-01 Effect of B CELL depletion by Rituximab in T cell subpopupulations from the peripheral blood of patients with rheumatoid arthritis576576, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2784]], camposKey:0022-3751 2012-01-01 High blood pressure associates with the remodelling of inward rectifier K plus channels in mice mesenteric vascular smooth muscle cells60756091, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2785]], camposKey:1079-5642 2012-01-01 Kv1.3 Channels Can Modulate Cell Proliferation During Phenotypic Switch by an Ion-Flux Independent Mechanism12991307, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=39767]], camposKey:0735-1097 2012-01-01 ROLE OF KV1.3 CHANNELS IN INTIMAL HYPERPLASIA00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2786]], camposKey:0008-6363 2010-01-01 Cell cycle-dependent expression of Kv3.4 channels modulates proliferation of human uterine artery smooth muscle cells383391, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2776]], camposKey:1079-5642 2010-01-01 Characterization of Ion Channels Involved in the Proliferative Response of Femoral Artery Smooth Muscle Cells12031211, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2770]], camposKey:0022-1295 2009-07-01 Specific Pattern of Ionic Channel Gene Expression Associated with Proliferation of Vascular Smooth Muscle Cells00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2779]], camposKey:0022-3751 2009-01-01 De novo expression of Kv6.3 contributes to changes in vascular smooth muscle cell excitability in a hypertensive mice strain625640, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2765]], camposKey:0065-2598 2009-01-01 DPPX Modifies TEA Sensitivity of the Kv4 Channels in Rabbit Carotid Body Chemoreceptor Cells7382, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=39769]], camposKey:0735-1097 2009-01-01 Functional Characterization of Potassium Channels and Their Role in the Modulation of Smooth Muscle Cell Phenotype in the Arterial Wall00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2761]], camposKey:0363-6143 2009-01-01 MaxiK potassium channels in the function of chemoreceptor cells of the rat carotid body00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=63342]], camposKey:0077-8923 2009-01-01 Oxygen-Sensitive Potassium Channels in Chemoreceptor Cell Physiology Making a Virtue of Necessity8288, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2757]], camposKey:0022-1295 2008-01-01 A role for DPPX modulating external TEA sensitivity of Kv4 channels455471, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2791]], camposKey:0300-8932 2008-01-01 The HERACLES cardiovascular network6675, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=74403]], camposKey:1885-5857 2008-01-01 The HERACLES cardiovascular network6675, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2750]], camposKey:0009-7330 2007-01-01 An ASIC channel for acid chemotransduction965967, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2754]], camposKey:1569-9048 2007-01-01 Oxygen sensitive Kv channels in the cartoid body6574, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2773]], camposKey:0363-6135 2006-01-01 Differential modulation of Kv4.2 and Kv4.3 channels by calmodulin-dependent protein kinase II in rat cardiac myocytes00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=851]], camposKey:0022-3751 2005-01-01 Comparative gene expression profile of mouse carotid body and adrenal medulla under physiological hypoxia491503, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2780]], camposKey:0009-7330 2005-01-01 Contribution of Kv channels to phenotypic remodeling of human uterine artery smooth muscle cells12801287, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2755]], camposKey:0022-3751 2005-01-01 Down regulation of Kv3.4 channels by chronic hypoxia increases acute oxygen sensitivity in rabbit carotid body395408, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2752]], camposKey:0022-3751 2004-01-01 Characterization of the Kv channels of mouse carotid body chemoreceptor cells and their role in oxygen sensing457471, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2744]], camposKey:0006-3495 2004-01-01 Hypoxic modulation of Kv channels in mouse carotid body cells00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=558]], camposKey:0022-3751 2004-01-01 Ventilatory responses and carotid body function in adult rats perinatally exposed to hyperoxia126144, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=41581]], camposKey:0065-2598 2003-01-01 Effects of Perinatal Hyperoxia on Carotid Body Chemoreceptor Activity in Vitro517524, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2749]], camposKey:0065-2598 2003-01-01 Functional identification of Kv alpha subunits contributing to the O-2-sensitive K+ current in rabbit carotid body chemoreceptor cells3339, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=63341]], camposKey:0210-0010 2002-04-16 Calcification of the thoracic yellow ligament as a cause of myelopathy750753, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=61744]], camposKey:0065-2598 2002-01-01 Effects of Perinatal Hyperoxia on Carotid Body Chemoreceptor Activity in Vitro517524, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=41724]], camposKey:0065-2598 2002-01-01 Functional Identification of Kv¿ Subunits Contributing to the O 2-Sensitive K+ Current in Rabbit Carotid Body Chemoreceptor Cells3339, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=41725]], camposKey:0022-3751 2002-01-01 Molecular identification of Kv alpha subunits that contribute to the oxygen-sensitive K+ current of chemoreceptor cells of the rabbit carotid body369382, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2739]], camposKey:0006-3495 2001-01-01 Ca2+-dependent K channels are modulated by hypoxia and CO in isolated membrane patches of rat chemoreceptor00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2768]], camposKey:0009-7330 2001-01-01 O-2 modulates large-conductance Ca2+-dependent K+ channels of rat chemoreceptor cells by a membrane-restricted and CO-sensitive mechanism430436, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2767]], camposKey:0009-7330 2000-01-01 Are Kv channels the essence of O-2 sensing?490491, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2751]], camposKey:0953-816X 2000-01-01 Functional significance of intracellular CA2+stores in rabbit carotid body chemoreceptor cells.5252, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2756]], camposKey:0363-6143 2000-01-01 Intracellular Ca2+ stores in chemoreceptor cells of the rabbit carotid body: significance for chemoreception5161, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2745]], camposKey:0953-816X 2000-01-01 Modulation of rat carotid body Maxi-K channels by hypoxia and carbon monoxide in isolated patches.2626, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2769]], camposKey:0270-6474 2000-01-01 Viral gene transfer of dominant-negative Kv4 construct suppresses an O-2-sensitive K+ current in chemoreceptor cells56895695, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2760]], camposKey:0022-1295 1999-01-01 Kv beta 1.2 subunit coexpression in HEK293 cells confers O-2 sensitivity to Kv4.2 but not to Shaker channels897907, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2753]], camposKey:0026-895X 1998-01-01 Effects of almitrine bismesylate on the ionic currents of chemoreceptor cells from the carotid body330339, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2747]], camposKey:0022-3751 1998-01-01 Kv beta 1 subunits can modulate the sensitivity to hypoxia of heterologously expressed voltage-gated K+ channels in a subfamily-specific manner00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2734]], camposKey:0031-6768 1997-01-01 Carbon monoxide inhibits hypoxic pulmonary vasoconstriction in rats by a cGMP-independent mechanism698704, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2764]], camposKey:0022-3751 1997-01-01 Properties of ionic currents from isolated adult rat carotid body chemoreceptor cells: Effect of hypoxia429441, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2758]], camposKey:0065-2598 1996-01-01 Intracellular Ca2+ deposits and catecholamine secretion by chemoreceptor cells of the rabbit carotid body279284, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=63344]], camposKey:0009-7322 1995-10-15 LOCAL CA2+-TRANSIENTS (CA2+-SPARKS) TRIGGERED BY SINGLE L-TYPE CALCIUM CURRENTS IN GUINEA-PIG HEART-CELLS20592059, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=63343]], camposKey:0300-5208 1995-01-01 CALCIUM SIGNALING IN CARDIAC-MUSCLE-CELLS146160, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2740]], camposKey:0300-5208 1995-01-01 Calcium signalling in cardiac muscle cells146164, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=63345]], camposKey:0300-5208 1995-01-01 Calcium signalling in cardiac muscle cells.00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2782]], camposKey:0034-5687 1995-01-01 Cellular mechanisms of oxygen chemoreception in the carotid body137147, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2736]], camposKey:0036-8075 1995-01-01 Local calcium transients triggered by single L-type calcium channel currents in cardiac cells10421045, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2737]], camposKey:0006-3495 1994-02-01 GAIN OF CICR IN CARDIAC-CELLS DEPENDS ON MEMBRANE VOLTAGE AND ON SR CA2+ CONTENT00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2763]], camposKey:0022-3751 1994-01-01 Inhibition of [3H]catecholamine release and Ca2+ currents by prostaglandin E2 in rabbit carotid body chemoreceptor cells.269277, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2748]], camposKey:0022-3751 1994-01-01 Local control of excitation¿contraction coupling in rat heart cells463471, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2746]], camposKey:0022-3751 1994-01-01 Local, stochastic release of Ca2+ in voltage¿clamped rat heart cells: visualization with confocal microscopy2129, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2766]], camposKey:0065-2598 1993-01-01 Ca2+ Dynamics in Chemoreceptor Cells: An Overview149156, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2742]], camposKey:0022-3751 1993-01-01 Properties of a transient K+ current in chemoreceptor cells of rabbit carotid body1532, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=38529]], camposKey:0002-9513 1992-01-01 Characterization of cultured chemoreceptor cells dissociated from adult rabbit carotid body00, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2738]], camposKey:0014-5793 1992-01-01 Time course of K+ current inhibition by low oxygen in chemoreceptor cells of adult rabbit carotid body Effects of carbon monoxide251254, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2735]], camposKey:0022-3751 1991-01-01 Release of dopamine and chemoreceptor discharge induced by low pH and high PCO2 stimulation of the cat carotid body.519531, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2774]], camposKey:0022-1295 1989-01-01 Ionic currents in dispersed chemoreceptor cells of the mammalian carotid body979999, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2772]], camposKey:0022-1295 1989-01-01 Low pO2 selectively inhibits k channel activity in chemoreceptor cells of the mammalian carotid body10011015, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=2771]], camposKey:0036-8075 1988-01-01 Chemotransduction in the carotid body: K+ current modulated by PO2 in type I chemoreceptor cells580582, com.sigma.fs3.argos.domain.gpc.GpcArticlesRev[id=com.sigma.fs3.argos.domain.gpc.GpcArticlesRevPK[ifcactivitat=ARE, ifccomptador=5554]], camposKey:0025-7753 1987-01-01 Reacciones adversas a medicamentos como urgencias externas en el Hospital Clínico Universitario de Valladolid130131][CapitolsLlib{id=com.sigma.fs3.argos.domain.gpc.GpcCapitolsLlibPK[ifcactivitat=CAP, ifccomptador=320]},camposKey: 82 88 Oxygen-Sensitive Potassium Channels in Chemoreceptor Cell Physiology Making a Virtue of Necessity 978-1-57331-773-3Hypoxia and Consequences 1 2009-11-23 -, CapitolsLlib{id=com.sigma.fs3.argos.domain.gpc.GpcCapitolsLlibPK[ifcactivitat=CAP, ifccomptador=93]},camposKey: 517 524 Effects of perinatal hyperoxia on carotid body chemoreceptor activity in vitro 0306478684Chemoreception 1 2003-10-31 -, CapitolsLlib{id=com.sigma.fs3.argos.domain.gpc.GpcCapitolsLlibPK[ifcactivitat=CAP, ifccomptador=1215]},camposKey: 663 678 Control de la ventilación pulmonar 8448602374Fisiología humana 2 1999-01-01 Interamericana-McGraw-Hill, CapitolsLlib{id=com.sigma.fs3.argos.domain.gpc.GpcCapitolsLlibPK[ifcactivitat=CAP, ifccomptador=1219]},camposKey: 69 87 Quimiorreceptores arteriales y centrales el control de las funciones respiratoria y circulatoria 8447204626Neurobiología de las funciones vegetativas 1 1998-01-01 Editorial Universidad de Sevilla , CapitolsLlib{id=com.sigma.fs3.argos.domain.gpc.GpcCapitolsLlibPK[ifcactivitat=CAP, ifccomptador=1216]},camposKey: 65 Electrical properties of carotid body chemoreceptor cells. Effects of natural stimuli 9783540619482The carotid body chemoreceptors 1 1997-01-01 Springer, CapitolsLlib{id=com.sigma.fs3.argos.domain.gpc.GpcCapitolsLlibPK[ifcactivitat=CAP, ifccomptador=319]},camposKey: 279 284 Intracellular Ca2+ deposits and catecholamine secretion by chemoreceptor cells of the rabbit carotid body 978-1-4613-7702-3Frontiers in Arterial Chemoreception 1 1996-01-01 Springer, CapitolsLlib{id=com.sigma.fs3.argos.domain.gpc.GpcCapitolsLlibPK[ifcactivitat=CAP, ifccomptador=14720]},camposKey: 44 57 Ionic Mechanisms of the Chemoreception Process in Type 1 Cells of the Carotid Body 0-387-97225-0Arterial Chemoreception 1 1990-08-06 -, CapitolsLlib{id=com.sigma.fs3.argos.domain.gpc.GpcCapitolsLlibPK[ifcactivitat=CAP, ifccomptador=25417]},camposKey: 44 57 IONIC MECHANISMS OF THE CHEMORECEPTION PROCESS IN TYPE-I CELLS OF THE CAROTID-BODY 0-387-97225-0Arterial Chemoreception 1 1990-08-06 -][][][][][][com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=64245]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=61762]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=63661]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=61893]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=62327]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=63631]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=12]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=13]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=14]]][com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=64245]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=61762]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=63661]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=61893]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=62327]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=63631]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=12]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=13]], com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBeca[id=com.sigma.fs3.argos.domain.gpc.GpcAltresAjutsBecaPK[ifcactivitat=AAB, ifccomptador=14]]][com.sigma.fs3.argos.domain.gpc.Premis[id=com.sigma.fs3.argos.domain.gpc.GpcPremisPK[ifcactivitat=PRE, ifccomptador=11]]][com.sigma.fs3.argos.domain.gpc.Premis[id=com.sigma.fs3.argos.domain.gpc.GpcPremisPK[ifcactivitat=PRE, ifccomptador=11]]][com.sigma.fs3.argos.domain.gpc.GpcTesis[id=com.sigma.fs3.argos.domain.gpc.GpcTesisPK[ifcactivitat=TES, ifccomptador=31145]], com.sigma.fs3.argos.domain.gpc.GpcTesis[id=com.sigma.fs3.argos.domain.gpc.GpcTesisPK[ifcactivitat=TES, ifccomptador=30216]], com.sigma.fs3.argos.domain.gpc.GpcTesis[id=com.sigma.fs3.argos.domain.gpc.GpcTesisPK[ifcactivitat=TES, ifccomptador=2145637418]], com.sigma.fs3.argos.domain.gpc.GpcTesis[id=com.sigma.fs3.argos.domain.gpc.GpcTesisPK[ifcactivitat=TES, ifccomptador=2145638388]], com.sigma.fs3.argos.domain.gpc.GpcTesis[id=com.sigma.fs3.argos.domain.gpc.GpcTesisPK[ifcactivitat=TES, ifccomptador=2145637678]], com.sigma.fs3.argos.domain.gpc.GpcTesis[id=com.sigma.fs3.argos.domain.gpc.GpcTesisPK[ifcactivitat=TES, ifccomptador=2145631436]], com.sigma.fs3.argos.domain.gpc.GpcTesis[id=com.sigma.fs3.argos.domain.gpc.GpcTesisPK[ifcactivitat=TES, ifccomptador=2145631521]], com.sigma.fs3.argos.domain.gpc.GpcTesis[id=com.sigma.fs3.argos.domain.gpc.GpcTesisPK[ifcactivitat=TES, ifccomptador=2145626732]], com.sigma.fs3.argos.domain.gpc.GpcTesis[id=com.sigma.fs3.argos.domain.gpc.GpcTesisPK[ifcactivitat=TES, ifccomptador=658]]][][][][][][][][com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@a3989f8, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@296df0fa, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@33b61130, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@6f264c11, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@29966e44, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@6f3aa1ac, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@6c0602ea, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@319d8dbf, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@2be33186, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@116ec297, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@4d239302, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@108e0f6c, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@22d373fa, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@454e674d, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@14245765, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@7431dfe2, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@4d443e3d, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@7b064cd7, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@4d2fa3ad, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@371bbb25, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@5bc2de2d, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@12eb4176, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@2269ba4d, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@4841ffb6, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@53f7325, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@4d976205, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@22c30228, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@6538e589, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@1b600450, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@6d11ca73, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@577947bb, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@754ff470, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@6e163c0d, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@d3721ee, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@10eefb6c, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@1a087175, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@4521c06d, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@71ada176, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@167f11b4, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@4f2549d7, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@44cdaf6, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@18767d18, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@766a3d13, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@13e4b9f8, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@719d4a18, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@5aa8778e, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@7d8d5b6d, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@4b90165d, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@504648bd][com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@82f919c, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@7f99080e, com.sigma.investigacion.cawdos.utilidades.CongresoYSusAsociaciones@52abf7c9][com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ConvenisPPC@5f69a8, com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ConvenisPPC@373e12][][][com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=60995]], com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=34]], com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=58035]], com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=35]], com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=36]]][com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=60995]], com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=34]], com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=58035]], com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=35]], com.sigma.fs3.argos.domain.gpc.EstadesFora[id=com.sigma.fs3.argos.domain.gpc.GpcEstadesForaPK[ifcactivitat=ESF, ifccomptador=36]]][com.sigma.fs3.argos.domain.gpc.GpcExpGestionId[id=com.sigma.fs3.argos.domain.gpc.GpcExpGestionIdPK[ifcactivitat=EXP, ifccomptador=31]], com.sigma.fs3.argos.domain.gpc.GpcExpGestionId[id=com.sigma.fs3.argos.domain.gpc.GpcExpGestionIdPK[ifcactivitat=EXP, ifccomptador=32]], com.sigma.fs3.argos.domain.gpc.GpcExpGestionId[id=com.sigma.fs3.argos.domain.gpc.GpcExpGestionIdPK[ifcactivitat=EXP, ifccomptador=33]], com.sigma.fs3.argos.domain.gpc.GpcExpGestionId[id=com.sigma.fs3.argos.domain.gpc.GpcExpGestionIdPK[ifcactivitat=EXP, ifccomptador=35]], com.sigma.fs3.argos.domain.gpc.GpcExpGestionId[id=com.sigma.fs3.argos.domain.gpc.GpcExpGestionIdPK[ifcactivitat=EXP, ifccomptador=34]], com.sigma.fs3.argos.domain.gpc.GpcExpGestionId[id=com.sigma.fs3.argos.domain.gpc.GpcExpGestionIdPK[ifcactivitat=EXP, ifccomptador=36]], com.sigma.fs3.argos.domain.gpc.GpcExpGestionId[id=com.sigma.fs3.argos.domain.gpc.GpcExpGestionIdPK[ifcactivitat=EXP, ifccomptador=37]]][com.sigma.fs3.argos.domain.gpc.GpcPatents[id=com.sigma.fs3.argos.domain.gpc.GpcPatentsPK[ifcactivitat=PAT, ifccomptador=2]]][][com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@467c0d], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@45f309], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@414d40], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@462806], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@413396], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@4125a9], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@41253c], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@413f06], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@413ff7], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@413db8], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@413c14], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@413fe4], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@413e90], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@412413], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@41200e], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@41235b], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@412124], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@412235], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@4122e6], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@4121f5], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@4605d3], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@411f75], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@46044c], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@411f0c], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@45f404], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@412255], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@45f45c], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@413ac9], com.sigma.investigacion.cawdos.entities.ayudaInvestigacion.ProjectesPPC[id=com.sigma.fs3.argos.domain.gpc.ayudasrecerca.ProjectesPPCId@413b55]][][][][][IftpertinEntitats[ iftpertinEntitatsPK=IftpertinEntitatsPK[ ifcactivitat=PEC, ifccomptador=27 ] ]][com.sigma.fs3.argos.domain.gpc.altres.ProjecteInnovacio@15f2e, com.sigma.fs3.argos.domain.gpc.altres.ProjecteInnovacio@15c28, com.sigma.fs3.argos.domain.gpc.altres.ProjecteInnovacio@1556b][]

PUBLICACIONES

Artículos de revista (96)

Serna J.; Peraza D.A.; Moreno-Estar S.; Saez J.J.; Gobelli D.; Simarro M.; Hivroz C.; Lopez-Lopez J.R.; Cidad P.; de la Fuente M.A.; Perez-Garcia M.T. Characterization of endogenous Kv1.3 channel isoforms in T cells. JOURNAL OF CELLULAR PHYSIOLOGY 2023; 0.

Enlace a la publicaciónTexto completo en acceso abierto
Ver Todos

Capítulos de libros (8)

Gonzalez, Constancio; Vaquero, Luis M.; Ramon Lopez-Lopez, Jose; Teresa Perez-Garcia, M. Oxygen-Sensitive Potassium Channels in Chemoreceptor Cell Physiology Making a Virtue of Necessity. En: -. Hypoxia and Consequences. -; 2009. p. 82-88.

Enlace a la publicación

Ver Todos
 

AYUDA A LA INVESTIGACIÓN

Proyectos (29)

CANALES IÓNICOS Y FISIOPATOLOGÍA VASCULAR: HISTORIA DE DOS CANALES. Equipo Investigadores: LOPEZ LOPEZ, JOSE RAMON (IP); PEREZ GARCIA, MARIA TERESA (IP); FUENTE GARCIA, MIGUEL ANGEL DE LA; SIMARRO GRANDE, MARIA. Equipo de Trabajo: GOBELLI, DINO JOAQUIN; DAGHBOUCHE RUBIO, NURIA; SERNA PÉREZ, JULIA; MORENO ESTAR, SARA; ROJO MENCIA, JORGE; MARRUGAT DE LA IGLESIA, JAUME; JAISSER ., FREDERIC; PERAZA PEREZ, DIEGO ALBERTO; REBOLLO MATO, IRINA. Personal Contratado: ALONSO ALONSO, ESPERANZA. PID2020-118517RB-I00. Entidades Participantes: UNIVERSIDAD DE VALLADOLID (UVa). Entidades Financiadoras: AGENCIA ESTATAL DE INVESTIGACIÓN, 10.13039/501100011033, MICINN. MINISTERIO DE CIENCIA E INNOVACIÓN . 01/09/2021-31/08/2024

Mnisterio de Ciencia e InnovaciónAEI
Ver Todos

Convenios/Contratos OTC (2)

INVESTIGACIÓN DEL PAPEL DEL KV1.3 EN LA FISIOPATOLOGÍA DE LAS ENFERMEDADES VASCULARES.. Equipo Investigadores: LOPEZ LOPEZ, JOSE RAMON (IP). Entidades Participantes: FUNDACION GENERAL DE LA UNIVERSIDAD DE VALLADOLID.. Entidades Financiadoras: JOSE RAMON LOPEZ LOPEZ., MARIA TERESA PEREZ GARCIA.. 22/09/2020

Ver Todos

Otras ayudas y becas (9)

UNIVERSIDAD DE VALLADOLID (UVa). CONTRATO PREDOCTORAL UVA DE REBOLLO MATO, IRINA: CANALES IÓNICOS Y FISIOPATOLOGÍA VASCULAR PROYECTO-SDP. LOPEZ LOPEZ, JOSE RAMON (Director). REBOLLO MATO, IRINA (Beneficiario). 01/05/2023 - 30/04/2027. 94.700,76 EUR.

Banco SantanderUniversidad de Valladolid
Ver Todos
 

OTROS

Tesis doctorales (9)

DAGHBOUCHE RUBIO, NURIA Remodeling of G protein coupled receptors signaling pathways in essential hypertension. Universidad de Valladolid; 2024

 
Ver Todos

Congresos (87)

Genetic & Animal Models for Ion Channel Fuction in Physiology & Disease, Woods Hole ( ESTADOS UNIDOS DE AMÉRICA ) 06/09/2016 - 11/09/2016
(Otros). Pérez-García, MT; Alvarez-Miguel, I; Cidad, P; López-López, JR; TRPC Channel Remodeling in a Mouse of Essential Hypertension: The TRPC3-TRPC6 Game of Thrones.
Ver Todos

Organización de actos (3)

Segunda Reunión Española de Canales Iónicos (RECI-II), 15/10/2009 - 16/10/2009
Otros.
Ver Todos

Premios (1)

López López, J.;. . Premio de investigación Garrrido Capa'. REAL ACADEMIA DE MEDICINA Y CIRUGIA DE VALLADOLID. 2007

Ver Todos

Patentes (1)

Pérez García, M.; López López, J.; Cidad, P.; Heras, M.; Roqué, M.; K channel blockers against restenosis. PCT/EP2009/063099. 2011
Ver Todos

Estancias de investigación (5)

ESTANCIA DE INVESTIGACIÓN SALVADOR MADARIAGA 2021 EN LUDC (LUND UNIVERSITY DIABETES CENTER). 01/04/2022 - 30/09/2022
     LUND UNIVERSITY. ( SUECIA )


Ver Todos

Experiencia en gestión de I+D (7)

Miembro de la Comisión de Desarrollo, Otros, 31/10/2006
Ver Todos

Entidades científicas (1)

. Otros.
Ver Todos

Proyectos de innovación docente (3)

Programa Orienta en el Grado en Ingeniería Biomédica en tiempos de Covid-19 - PID2020/060. Universidad de Valladolid. 01/09/2020.
Ver Todos
Cargando información ...
Universidad de Valladolid - Copyright Universidad de Valladolid - Todos los derechos reservados